Researchers Link Specific Protein Mutations to Ataxia Disease Symptoms November 7, 2019 By vdoyle showing that limiting the activity of a mutant CHIP protein could decrease symptom severity for people with cerebral ataxia, a debilitating disease of the nervous system. Filed Under: Categories: News More from Computational Medicine Dr. Elizabeth Brunk for being awarded the 2025 PhRMA Foundation Faculty Brunk Lab | CytoCellDB WORKSHOP | the Gaussian Accelerated Molecular Dynamics (GaMD)
showing that limiting the activity of a mutant CHIP protein could decrease symptom severity for people with cerebral ataxia, a debilitating disease of the nervous system.